Saturday, November 21, 2020

Cholesterol Levels:"Good" cholesterol VS "Bad" cholesterol

Cholesterol Levels:"Good" cholesterol VS "Bad" cholesterol

Over the past two decades, a controversy has emerged among scientists about the role of cholesterol in the cause of cardiovascular disease (CVD). This dispute, which has often taken on a bitter tone, is still ongoing and so far has not produced positive results for patients. In my opinion, high cholesterol levels are more a symptom of reduced circulation than a factor causing CVD. This is certainly the condition where there are high cholesterol levels along with other pathological changes and where all these abnormalities can be counteracted by an anabolic factor. Therefore, I am against those scientists who believe that a higher cholesterol level is the cause of cardiovascular disease

Cholesterol Levels 

There are several types of lipoproteins. The largest is called chylomicron, not lipoprotein. A chylomicron is a form of lipoprotein, this particular lipoprotein is produced in the intestine. After a meal, it is filled with triglycerides (fats), along with a relatively small amount of cholesterol. Chylomicron then travels directly to the body's fat cells without passing through the liver. When chylomicron reaches the fat cells, the triglyceride is absorbed and the chylomicron shrinks into a small wilted residue that is likely suspended from the liver

A lower level of size from a chylomicron is a lipoprotein known as a very low density lipoprotein (VLDL). VLDL are produced in both the intestine and liver. Just to add even more confusion to this area, VLDLs are generally referred to as "triglycerides - some of you may have even been informed of your triglyceride level (this is something relatively new that affects people)

Another smaller level is intermediate density lipoprotein (IDL). This is formed when a VLDL loses triglycerides in fat cells and shrinks. When IDL is reduced to an even lower level, it is converted into low density lipoprotein (LDL)

Total cholesterol: this is probably the most common number given to patients. The total cholesterol level is achieved by adding the "bad" LDL levels, plus the "good" HDL, as well as some other rebellious lipoproteins, IDL and the like, which are mixed in the analysis

• A chylomicron is a lipoprotein, but it is never called that

• A VLDL is a lipoprotein, but it is generally called triglyceride

• An LDL is a lipoprotein but is called "bad" cholesterol

• A high density lipoprotein (HDL) is smallest lipoprotein, is called "good" cholesterol

 "Good" cholesterol

This is the level of high density lipoprotein (HDL), which is normally around 1.3 mmol / l. Anything below O.9mmolll and your life insurance company will cancel the subscription, then paint a small black dot in the center of the palm for good luck. Above 2.0 mmol / l, you can get the saga brochures and wait for a long and happy career in green bowling

HDL is basically the opposite of LDL. Instead of being high in fat, HDL is high in protein. Instead of carrying cholesterol in the body, HDL acts like a vacuum cleaner that absorbs cholesterol as much as possible. It collects extra cholesterol from cells and tissues and returns it to the liver, which extracts cholesterol from the particle and uses it to produce or recycle bile. This action is believed to explain why high HDL levels are associated with a low risk of heart disease. HDL also contains antioxidant molecules that can prevent LDL from becoming a lipoprotein that is even more prone to cause heart disease

"Bad" cholesterol

level This is the level of low density lipoprotein (LDL). On average, this is around 3.5 mmol / l. Something above 4mmol / l will be classified. Resistance is futile

LDL is clearly the most important proatherogenic lipoprotein. It is the main lipoprotein containing cholesterol. It is the only markedly elevated fraction in familial hypercholesterolaemia. It is  probability, the most commonly elevated lipoprotein fraction in the hypercholesterolaemic patient  at high risk of coronary heart disease. Current therapy is primarily driven by the response of LDL levels to intervention

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